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Carbohydrate Restriction-Induced Elevations in LDL-Cholesterol and Atherosclerosis: The KETO Trial | JACC: Advances
Coronary plaque in metabolically healthy individuals with carbohydrate restriction-induced LDL-C ≥190 mg/dL on KETO for a mean of 4.7 years is not greater than a matched cohort with 149 mg/dL lower average LDL-C. There is no association between LDL-C and plaque burden in either cohort. (Diet-induced Elevations in LDL-C and Progression of Atherosclerosis [Keto-CTA]; NCT057333255)
How this Guy Cut His Cholesterol in Half Without Drugs and Got Ripped
No one could explain why my LDL cholesterol was so high. The most logical explanation—a condition called familial hypercholesterolemia (FH)—wasn’t comforting: It was genetics, and research shows that people with very high LDL and FH have a 22 times greater risk of developing coronary artery disease.
My triglycerides are very low
Triglycerides are a type of fat (lipid) found in your blood.
When you eat, your body converts any calories it doesn't need to use right away into triglycerides. The triglycerides are stored in your fat cells. Later, hormones release triglycerides for energy between meals.
If you regularly eat more calories than you burn, particularly from high-carbohydrate foods, you may have high triglycerides (hypertriglyceridemia).
What's considered normal?
A simple blood test can reveal whether your triglycerides fall into a healthy range:
Normal — Less than 150 milligrams per deciliter (mg/dL), or less than 1.7 millimoles per liter (mmol/L)
My non-HDL is below 130
An optimal level of non-HDL cholesterol for most people is less than 130 milligrams per deciliter (mg/dL), which is 3.37 millimoles per liter (mmol/L). For people with a history of heart attack, the desired level may be lower. Higher numbers mean a higher risk of heart disease.
Lipoprotein (a) Meaning and How Does it Impact My Heart Health? | American Heart Association
High Lp(a) numbers of 50 mg/dL (125 nmols/L) or higher promote clotting and inflammation, significantly increasing risk of heart attack, stroke, aortic stenosis and peripheral artery disease. This is especially true for those with coronary heart disease or familial hypercholesterolemia, or FH, an inherited condition that affects the body’s ability to process LDL “bad” cholesterol.
Lp(a) can accumulate in the walls of blood vessels, forming plaques similarly to LDL cholesterol. These plaques can block blood flow to vital organs such as the heart, brain, kidneys, lungs, and other parts of the body, leading to conditions like heart attacks, strokes, and other cardiovascular diseases.
